1. |
Fisher RS, Acevedo C, Arzimanoglou A, et al. ILAE official report: a practical clinical definition of epilepsy. Epilepsia, 2014, 55(4): 475-482.
|
2. |
Chang BS, Lowenstein DH. Epilepsy. N Engl J Med, 2003, 349(13): 1257-1266.
|
3. |
Margerison JH, Corsellis JA. Epilepsy and the temporal lobes. A clinical, electroencephalographic and neuropathological study of the brain in epilepsy, with particular reference to the temporal lobes. Brain, 1966, 89(3): 499-530.
|
4. |
Thurman DJ, Beghi E, Begley CE, et al. Standards for epidemiologic studies and surveillance of epilepsy. Epilepsia, 2011, 52(Suppl 7): 2-26.
|
5. |
De Lanerolle NC, Kim JH, Williamson A, et al. A retrospective analysis of hippocampal pathology in human temporal lobe epilepsy: evidence for distinctive patient subcategories. Epilepsia, 2003, 44(5): 677-687.
|
6. |
Spencer SS, Spencer DD. Entorhinal‐hippocampal interactions in medial temporal lobe epilepsy. Epilepsia, 1994, 35(4): 721-727.
|
7. |
Lévesque M, Avoli M. The kainic acid model of temporal lobe epilepsy. Neurosci Biobehav Rev, 2013, 37(10 Pt2): 2887-2899.
|
8. |
Murakami S, Takemoto T. On the effective principles of digenea‐simplex aq. 1. Separation of the effective fraction by liquid chromatography. Yakugaku, 1953.
|
9. |
Watkins JC, Evans RH. Excitatory amino acid transmitters. Annu Rev Pharmacol Toxicol, 1981, 21: 165-204.
|
10. |
Ben‐Ari Y, Lagowska J. Epileptogenic action of intra‐amygdaloid injection of kainic acid. C R Acad Sci Hebd Seances Acad Sci D, 1978, 287(8): 813-816.
|
11. |
Ben‐Ari Y, Lagowska J, Tremblay E, et al. A new model of focal status epilepticus: intra‐amygdaloid application of kainic acid elicits repetitive secondarily generalized convulsive seizures. Brain Res, 1979, 163(1): 176-179.
|
12. |
Lévesque M, Avoli M, Bernard C. Animal models of temporal lobe epilepsy following systemic chemoconvulsant administration. J Neurosci Methods, 2015, 260: 45-52.
|
13. |
Hellier JL, Dudek FE. Chemoconvulsant model of chronic spontaneous seizures. Curr Protoc Neurosci, 2005.
|
14. |
Alldredge BK, Gelb AM, Isaacs SM, et al. A comparison of lorazepam, diazepam, and placebo for the treatment of out‐of‐hospital status epilepticus. N Engl J Med, 2001, 345(9): 631-637.
|
15. |
Norwood BA, Bumanglag AV, Osculati F, et al. Classic hippocampal sclerosis and hippocampal‐onset epilepsy produced by a single " cryptic” episode of focal hippocampal excitation in awake rats. J Comp Neurol, 2010, 518(16): 3381-3407.
|
16. |
Harvey BD, Sloviter RS. Hippocampal granule cell activity and c‐Fos expression during spontaneous seizures in awake, chronically epileptic, pilocarpine‐treated rats: implications for hippocampal epileptogenesis. J Comp Neurol, 2005, 488(4): 442-463.
|
17. |
Racine RJ. Modification of seizure activity by electrical stimulation: III. Motor seizure. Electroencephalogr. Clin Neurophysiol, 1972, 32(3): 281-294.
|
18. |
Blümcke I, Thom M, Aronica E, et al. International consensus classification of hippocampal sclerosis in temporal lobe epilepsy: a Task Force report from the ILAE Commission on Diagnostic Methods. Epilepsia, 2013, 54(7): 1315-1329.
|
19. |
Trinka E, Cock H, Hesdorffer D, et al. A definition and classification of status epilepticus - report of the ILAE Task Force on Classification of Status Epilepticus. Epilepsia, 2015, 56(10): 1515-1523.
|
20. |
Williams PA, White AM, Clark S, et al. Development of spontaneous recurrent seizures after kainate‐induced status epilepticus. J Neurosci, 2009, 29(7): 2103-2112.
|
21. |
Deller T, Frotscher M, Nitsch R. Morphological evidence for the sprouting of inhibitory commissural fibers in response to the lesion of the excitatory entorhinal input to the rat dentate gyrus. J Neurosci, 1995, 15(10): 6868-6878.
|
22. |
Elger CE, Schmidt D. Modern management of epilepsy: a practical approach. Epilepsy Behav, 2008, 12(4): 501-539.
|
23. |
Löscher W, Schmidt D. Modern antiepileptic drug development has failed to deliver: ways out of the current dilemma. Epilepsia, 2011, 52(4): 657-678.
|
24. |
Arabadzisz D, Antal K, Parpan F, et al. Epileptogenesis and chronic seizures in a mouse model of temporal lobe epilepsy are associated with distinct EEG patterns and selective neurochemical alterations in the contralateral hippocampus. Exp Neurol, 2005, 194(1): 76-90.
|
25. |
Carriero G, Arcieri S, Cattalini A, et al. A guinea pig model of mesial temporal lobe epilepsy following nonconvulsive status epilepticus induced by unilateral intrahippocampal injection of kainic acid. Epilepsia, 2012, 53(11): 1917-1927.
|
26. |
Daniels WMU, Jaffer A, Engelbrecht AH, et al. The effect of intrahippocampal injection of kainic acid on corticosterone release in rats. Neurochem Res, 1990, 15(5): 495-499.
|
27. |
Victor Nadler J, Cuthbertson GJ. Kainic acid neurotoxicity toward hippocampal formation: dependence on specific excitatory pathways. Brain Res, 1980, 195(1): 47-56.
|
28. |
Raedt R, Van Dycke A, Van Melkebeke D, et al. Seizures in the intrahippocampal kainic acid epilepsy model: characterization using long‐term video‐EEG monitoring in the rat. Acta Neurol Scand, 2009, 119(5): 293-303.
|
29. |
Araki T, Simon RP, Taki W, et al. Characterization of neuronal death induced by focally evoked limbic seizures in the C57BL/6 mouse. J Neurosci Res, 2002, 69(5): 614-621.
|
30. |
Ben‐Ari Y, Tremblay E, Ottersen OP. Injections of kainic acid into the amygdaloid complex of the rat: an electrographic, clinical and histological study in relation to the pathology of epilepsy. Neuroscience, 1980, 5(3): 515-528.
|
31. |
Cavalheiro EA, Riche DA, Le Gal La Salle G. Long‐term effects of intrahippocampal kainic acid injection in rats: a method for inducing spontaneous recurrent seizures. Electroencephalogr Clin Neurophysiol, 1982, 53(6): 581-589.
|
32. |
Dunleavy M, Shinoda S, Schindler C, et al. Experimental neonatal status epilepticus and the development of temporal lobe epilepsy with unilateral hippocampal sclerosis. Am J Pathol, 2010, 176(1): 330-342.
|
33. |
Gurbanova AA, Aker RG, Sirvanci S, et al. Intra‐amygdaloid injection of kainic acid in rats with genetic absence epilepsy: the relationship of typical absence epilepsy and temporal lobe epilepsy. J Neurosci, 2008, 28(31): 7828-7836.
|
34. |
Jimenez‐Mateos EM, Engel T, Merino‐Serrais P, et al. Silencing microRNA‐134 produces neuroprotective and prolonged seizure‐suppressive effects. Nat Med, 2012, 18: 1087-1094.
|
35. |
Mouri G, Jimenez‐Mateos E, Engel T, et al. Unilateral hippocampal CA3‐predominant damage and short latency epileptogenesis after intra‐amygdala microinjection of kainic acid in mice. Brain Res, 2008, 1213: 140-151.
|
36. |
Represa A, Tremblay E, Ben‐Ari Y. Kainate binding sites in the hippocampal mossy fibers: localization and plasticity. Neuroscience, 1987, 20(3): 739-748.
|
37. |
Drexel M, Preidt AP, Sperk G. Sequel of spontaneous seizures after kainic acid‐induced status epilepticus and associated neuropathological changes in the subiculum and entorhinal cortex. Neuropharmacology, 2012, 63(5): 806-817.
|
38. |
Haas KZ, Sperber EF, Opanashuk LA, et al. Resistance of immature hippocampus to morphologic and physiologic alterations following status epilepticus or kindling. Hippocampus, 2001, 11(6): 615-625.
|
39. |
Heggli DE, Malthesorenssen D. Systemic injection of kainic acid ‐ effect on neurotransmitter markers in piriform cortex, amygdaloid complex and hippocampus and protection by cortical lesioning and anticonvulsants. Neuroscience, 1982, 7(5): 1257-1264.
|
40. |
Kar S, Seto D, Doré S, et al. Systemic administration of kainic acid induces selective time dependent decrease in[125I]insulin‐like growth factor I, [125I]insulin‐like growth factor II and[125I]insulin receptor binding sites in adult rat hippocampal formation. Neuroscience, 1997, 80(4): 1041-1055.
|
41. |
Sperk G, Lassmann H, Baran H, et al. Kainic acid‐induced seizures − dose‐relationship of behavioral, neurochemical and histopathological changes. Brain Res, 1985, 338: 289-295.
|
42. |
Sloviter RS, Damiano BP. Sustained electrical stimulation of the perforant path duplicates kainate‐induced electrophysiological effects and hippocampal damage in rats. Neurosci Lett, 1981, 24(3): 279-284.
|
43. |
Strain SM, Tasker R. Hippocampal damage produced by systemic injections of domoic acid in mice. Neuroscience, 1991, 44(2): 343-352.
|
44. |
Suarez LM, Cid E, Gal B, et al. Systemic injection of kainic acid differently affects LTP magnitude depending on its epileptogenic efficiency. PLoS ONE, 2012, 7(10): e48128.
|
45. |
Zhang X, Cui SS, Wallace AE, et al. Relations between brain pathology and temporal lobe epilepsy. J Neurosci, 2002, 22(14): 6052-6061.
|
46. |
Hellier JL, Patrylo PR, Buckmaster PS, et al. Recurrent spontaneous motor seizures after repeated low‐dose systemic treatment with kainate: assessment of a rat model of temporal lobe epilepsy. Epilepsy Res, 1998, 31(1): 73-84.
|