胰岛移植后移植物损伤的免疫学特点_CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY

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肖毅 , 何晓东 , 综述 , 朱预 , 审校

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Citation： 肖毅,何晓东,综述,朱预,审校. 胰岛移植后移植物损伤的免疫学特点. CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY, 1996, 3(1): 50-52. doi: Copy

1.	Thorsby E.The HLA molecules:function and role in allorecognition.Transplant Proc,1994;26:1699.
2.	Osorio RW,Ascher NL,Jaenisch R,et al.Major histocompatibility complex class Ⅰ deficiency prolongs islet allograft survival.Diabetes,1993;42:1520.
3.	Markmann JF,Tomaszewski J,Posselt AM,et al.The effect of islet cell culture in vitro at 24℃ on graft survival and MHC antigen expression.Transplanation,1990;49:272.
4.	Hao L,Wang Y,Gill RG,et al.Role of γ-interferoh in islet allograft rejection:class IMHC antigen induction.Transplant Proc,1989;21:2697.
5.	Hall BM.Cells mediating allograft rejection.Transplantation,1991;51:1141.
6.	Lafferty KJ,Hao L.Approaches to the prevention of immune destruction of transplanted pancreatic islets.Transplant Proc,1994;26:399.
7.	Mason DW,Simmonds SJ.The autonomy of CD8+T cells in vitro and in vivo.Immunolgy,1988;65:249.
8.	Bolton EM,Gracie JA,Briggs JD,et al.Cellular reqirements for renal allograft rejection in the athymic nude rat.J Exp Med,1989;169:1931.
9.	Yamashita Y,Bullington R,Clement LT.Equivalent helper functions of human"naive"and"memory"CD4+T cells for the generation of alloreactive cytotoxic T lymphocytes.J Clin Immunol,1990;10:237.
10.	Miller BJ.Appel MC,O’Nell JJ,et al.Both the Lyt-2+ and L3T4+T cell subsets are required for the transfer of diabetes in nonobese diabetic mice.J Immunol,1988;140:52.
11.	Charlton B,Mandel TE.Progression from insulitis to cell destruction in NOD mouse requires L3T4+T-lymphocytes.Diabetes,1988;37:1108.
12.	Wang Y,Pontesilli O,Gill RG,et al.The role of CD4+ and CD8+ T cells in the destruction of islet graft by spontaneouslydiabetic mice.Proc Natl Acad Sci USA,1991;88:527.
13.	Bradley BJ,Haskins K,LaRosa FG,et al.CD8+T cell are not required for islet destruction induced by a CD4+ islet specific T-cell clone.Diabetes,1992;41:1603.
14.	Rabinovitch A,Sumoski W,Rajotte RV,et al.Cytotoxic effects of cytokines on human pancreatic islet cells in monolayer culture.J Clin Endocrinol Metab,1990;71:152.
15.	Christianson SW,Shultz LD,Leiter EH.Adoptive transfer of diabetes intoimmunodeficient NOD-scid/scidmice:ralative contributions of CD4+ and CD8+ T-cell from diabetes versus prediabetic NOD.NON-Thy-la donors.Diabetes,1993;42:44.
16.	Ling Z,In’t Veld PA,Pipeleers DG.Interaction of interleukin-1 with islet β-cells:distinction between indirect,aspecific cytotoxicity and direct specific functional supperssion.Diabetes,1993;42:56.
17.	Hao L,Wang Y,Gill RG,et al.Role of lymphokine in islet allograft rjection.Transplantation,1990;49:609.

1. Thorsby E.The HLA molecules:function and role in allorecognition.Transplant Proc,1994;26:1699.
2. Osorio RW,Ascher NL,Jaenisch R,et al.Major histocompatibility complex class Ⅰ deficiency prolongs islet allograft survival.Diabetes,1993;42:1520.
3. Markmann JF,Tomaszewski J,Posselt AM,et al.The effect of islet cell culture in vitro at 24℃ on graft survival and MHC antigen expression.Transplanation,1990;49:272.
4. Hao L,Wang Y,Gill RG,et al.Role of γ-interferoh in islet allograft rejection:class IMHC antigen induction.Transplant Proc,1989;21:2697.
5. Hall BM.Cells mediating allograft rejection.Transplantation,1991;51:1141.
6. Lafferty KJ,Hao L.Approaches to the prevention of immune destruction of transplanted pancreatic islets.Transplant Proc,1994;26:399.
7. Mason DW,Simmonds SJ.The autonomy of CD8+T cells in vitro and in vivo.Immunolgy,1988;65:249.
8. Bolton EM,Gracie JA,Briggs JD,et al.Cellular reqirements for renal allograft rejection in the athymic nude rat.J Exp Med,1989;169:1931.
9. Yamashita Y,Bullington R,Clement LT.Equivalent helper functions of human"naive"and"memory"CD4+T cells for the generation of alloreactive cytotoxic T lymphocytes.J Clin Immunol,1990;10:237.
10. Miller BJ.Appel MC,O’Nell JJ,et al.Both the Lyt-2+ and L3T4+T cell subsets are required for the transfer of diabetes in nonobese diabetic mice.J Immunol,1988;140:52.
11. Charlton B,Mandel TE.Progression from insulitis to cell destruction in NOD mouse requires L3T4+T-lymphocytes.Diabetes,1988;37:1108.
12. Wang Y,Pontesilli O,Gill RG,et al.The role of CD4+ and CD8+ T cells in the destruction of islet graft by spontaneouslydiabetic mice.Proc Natl Acad Sci USA,1991;88:527.
13. Bradley BJ,Haskins K,LaRosa FG,et al.CD8+T cell are not required for islet destruction induced by a CD4+ islet specific T-cell clone.Diabetes,1992;41:1603.
14. Rabinovitch A,Sumoski W,Rajotte RV,et al.Cytotoxic effects of cytokines on human pancreatic islet cells in monolayer culture.J Clin Endocrinol Metab,1990;71:152.
15. Christianson SW,Shultz LD,Leiter EH.Adoptive transfer of diabetes intoimmunodeficient NOD-scid/scidmice:ralative contributions of CD4+ and CD8+ T-cell from diabetes versus prediabetic NOD.NON-Thy-la donors.Diabetes,1993;42:44.
16. Ling Z,In’t Veld PA,Pipeleers DG.Interaction of interleukin-1 with islet β-cells:distinction between indirect,aspecific cytotoxicity and direct specific functional supperssion.Diabetes,1993;42:56.
17. Hao L,Wang Y,Gill RG,et al.Role of lymphokine in islet allograft rjection.Transplantation,1990;49:609.

CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY

胰岛移植后移植物损伤的免疫学特点

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