Pathogenesis of Immune Dysfunction in Surgical Infection_CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY

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张庆红 , 姚咏明

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Keywords：

外科; 感染; 脓毒症; 免疫; 机理

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Citation： 张庆红,姚咏明. Pathogenesis of Immune Dysfunction in Surgical Infection. CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY, 2010, 17(11): 1129-1131. doi: Copy

1.	Fry DE, Fry RV. Surgical site infection: the host factor [J]. AORN J, 2007; 86(5): 801-810.
2.	Vogel TR, Dombrovskiy VY, Lowry SF. Trends in postoperative sepsis: are we improving outcomes? [J]. Surg Infect (Larchmt), 2009; 10(1): 71-78.
3.	姚咏明. 创伤感染并发症免疫功能障碍及其诊治的若干问题 [J]. 中华外科杂志, 2009; 47(1): 37-39.
4.	Fishman JA. Infection in solid-organ transplant recipients [J]. N Engl J Med, 2007; 357(25): 2601-2614.
5.	Lowry SF. The stressed host response to infection: the disruptive signals and rhythms of systemic inflammation [J]. Surg Clin North Am, 2009; 89(2): 311-326, vii.
6.	Cevenini E, Caruso C, Candore G, et al. Age-related inflammation: the contribution of different organs, tissues and systems. How to face it for therapeutic approaches [J]. Curr Pharm Des, 2010; 16(6): 609-618.
7.	Gameiro C, Romao F. Changes in the immune system during menopause and aging [J]. Front Biosci (Elite Ed), 2010; 2: 1299-1303.
8.	Raju R, Bland KI, Chaudry IH. Estrogen: a novel therapeutic adjunct for the treatment of trauma-hemorrhage-induced immunological alterations [J]. Mol Med, 2008; 14(3-4): 213-221.
9.	Villar J, Maca-Meyer N, Pérez-Méndez L, et al. Bench-to-bedside review: understanding genetic predisposition to sepsis [J]. Crit Care, 2004; 8(3): 180-189.
10.	Hughes SM, Amadi B, Mwiya M, et al. Dendritic cell anergy results from endotoxemia in severe malnutrition [J]. J Immunol, 2009; 183(4): 2818-2826.
11.	Gabrilovich DI, Nagaraj S. Myeloid-derived suppressor cells as regulators of the immune system [J]. Nat Rev Immunol, 2009; 9(3): 162-174.
12.	Ribechini E, Greifenberg V, Sandwick S, et al. Subsets, expansion and activation of myeloid-derived suppressor cells [J]. Med Microbiol Immunol, 2010; 199(3): 273-281.
13.	Gaestel M, Kotlyarov A, Kracht M. Targeting innate immunity protein kinase signalling in inflammation [J]. Nat Rev Drug Discov,.

1. Fry DE, Fry RV. Surgical site infection: the host factor [J]. AORN J, 2007; 86(5): 801-810.
2. Vogel TR, Dombrovskiy VY, Lowry SF. Trends in postoperative sepsis: are we improving outcomes? [J]. Surg Infect (Larchmt), 2009; 10(1): 71-78.
3. 姚咏明. 创伤感染并发症免疫功能障碍及其诊治的若干问题 [J]. 中华外科杂志, 2009; 47(1): 37-39.
4. Fishman JA. Infection in solid-organ transplant recipients [J]. N Engl J Med, 2007; 357(25): 2601-2614.
5. Lowry SF. The stressed host response to infection: the disruptive signals and rhythms of systemic inflammation [J]. Surg Clin North Am, 2009; 89(2): 311-326, vii.
6. Cevenini E, Caruso C, Candore G, et al. Age-related inflammation: the contribution of different organs, tissues and systems. How to face it for therapeutic approaches [J]. Curr Pharm Des, 2010; 16(6): 609-618.
7. Gameiro C, Romao F. Changes in the immune system during menopause and aging [J]. Front Biosci (Elite Ed), 2010; 2: 1299-1303.
8. Raju R, Bland KI, Chaudry IH. Estrogen: a novel therapeutic adjunct for the treatment of trauma-hemorrhage-induced immunological alterations [J]. Mol Med, 2008; 14(3-4): 213-221.
9. Villar J, Maca-Meyer N, Pérez-Méndez L, et al. Bench-to-bedside review: understanding genetic predisposition to sepsis [J]. Crit Care, 2004; 8(3): 180-189.
10. Hughes SM, Amadi B, Mwiya M, et al. Dendritic cell anergy results from endotoxemia in severe malnutrition [J]. J Immunol, 2009; 183(4): 2818-2826.
11. Gabrilovich DI, Nagaraj S. Myeloid-derived suppressor cells as regulators of the immune system [J]. Nat Rev Immunol, 2009; 9(3): 162-174.
12. Ribechini E, Greifenberg V, Sandwick S, et al. Subsets, expansion and activation of myeloid-derived suppressor cells [J]. Med Microbiol Immunol, 2010; 199(3): 273-281.
13. Gaestel M, Kotlyarov A, Kracht M. Targeting innate immunity protein kinase signalling in inflammation [J]. Nat Rev Drug Discov,.

CHINESE JOURNAL OF BASES AND CLINICS IN GENERAL SURGERY

Pathogenesis of Immune Dysfunction in Surgical Infection

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