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find Author "于晴" 2 results
  • 严重感染患者外周血中树突状细胞的变化及意义

    严重感染及感染性休克是全身性感染( sepsis) 导致的以器官功能损害为特征的复杂临床综合征, 在ICU患者的发病率高达25% [1] 。近年来, 尽管抗感染治疗及器官功能支持技术取得了长足的进步, 但严重感染的病死率仍高达30% ~70% [2] 。严重感染的发病机制复杂, 免疫功能的紊乱参与严重感染的发生与发展。树突状细胞( DCs) 在启动机体免疫应答中发挥关键性作用。近年研究显示, 严重感染患者外周血DCs 数量及功能呈现明显动态改变, 并与病情严重程度及预后相关。因此监测严重感染的危重病患者外周血DCs改变具有重要的临床意义。

    Release date:2016-08-30 11:54 Export PDF Favorites Scan
  • Correlation of the prothrombin gene G20210A mutation and cerebral venous thrombosis risk: a meta-analysis

    Objective To systematically review the association between prothrombin gene G20210A mutation and the risk of cerebral venous thrombosis (CVT). Methods Databases including PubMed, Springer, Google Scholar, The Cochrane Library (Issue 1, 2016), CNKI, WanFang Data and CBM were searched for case-control studies concerning the association between prothrombin gene G20210A mutation and cerebral venous thrombosis risk from inception to January 2016. Two reviewers independently screened literature, extracted data and assessed the risk of bias of included studies. Then, meta-analysis was performed using RevMan 5.3 software and Stata 12.0 software. Results A total of 26 case-control studies were included, involving 1 361 CVT cases and 6 323 controls. The results of meta-analysis showed that: there was a significant association between prothrombin gene G20210A mutation and CVT risk (OR=4.56, 95% CI 3.51 to 5.93,P<0.000 01). Sensitivity analysis showed no significant publication bias was detected confirmed the stability of results. Subgroup analysis showed that G20210A mutation increased CVT risk in adults (OR=5.02, 95% CI 3.81 to 6.60,P<0.000 01), but not in children (OR=1.99, 95% CI 0.83 to 4.79,P=0.12). Conclusion Prothrombin gene G20210A mutation can significantly increase the CVT risk. Due to the limited quality and quantity of included studies, the above results are needed to be validated by more high quality studies.

    Release date:2017-04-01 08:56 Export PDF Favorites Scan
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