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find Keyword "病理机制" 8 results
  • 睡眠呼吸暂停致缺血性脑卒中病理机制的研究进展

    睡眠呼吸暂停综合症被定义为7 h 睡眠中呼吸暂停及低通气反复发作在30 次以上, 或呼吸紊乱指数( AHI) ≥5 次/h。由其引起的睡眠时反复发生的低氧血症和高碳酸血症, 不仅可导致呼吸系统的病理生理变化, 同时可引起循环系统、血液系统、内分泌系统及神经系统等一系列连锁反应, 亦可引起包括缺血性脑卒中在内的一些危及生命的危重症[ 1 ] 。缺血性脑卒中是指突然发生的脑组织局部供血动脉血流灌注减少或血流完全中断, 停止供血、供氧、供糖等, 使该局部脑组织崩解破坏。Basetti 等[ 2] 研究发现, 在脑卒中患者中, 阻塞性睡眠呼吸暂停综合征 ( OSAS) 的患病率为44% ~72% , 而在正常人群中仅为4% 。而Fischer 等[ 3 ] 发现OSAS 患者发生脑梗死的概率是无OSAS 的31 倍。一项大型的前瞻性流行病学研究显示, 在校正其他危险因素( 包括高血压) 后, OSAS患者发生脑卒中或出现死亡的危险度为1. 97( 95% CI 1. 12 ~3. 48, P = 0. 02) [ 4 ] 。Arzt 等[ 5] 对1475 例研究对象的研究结果显示, 在校正年龄、性别、体重指数( BMI) 、吸烟及饮酒史等因素的影响后, 有重度OSAS者( AHI gt; 20 次/h) 较无OSAS 者( AHI lt; 5 次/h) 脑卒中发生有明显差异( OR 4. 33; 95% CI 1. 32 ~14. 24, P = 0. 02) , 从而证实了睡眠呼吸暂停综合征为脑卒中发生的独立危险因素。但其发生发展的具体机制尚不完全清楚。从目前国内外的多项研究推测, 可能与下列因素有关。

    Release date:2016-09-13 03:51 Export PDF Favorites Scan
  • 电刺激癫痫动物模型的研究进展

    癫痫以脑神经元异常放电引起反复痫性发作为特征。建立癫痫动物模型对研究癫痫的发病机制及治疗具有重要意义。近年来建立了多种动物模型, 主要包括化学点燃和电点燃模型等。经典的癫痫电点燃是对大脑边缘结构进行重复的电刺激, 导致逐渐增强的后放电和行为学上的癫痫发作。电点燃形成后, 即使不再给予刺激, 异常的痫性放电可以持续很长时间, 甚至终生。电刺激癫痫动物模型具有诱导致痫的优点, 而且与人类癫痫发生和形成极为相似。现就电点燃模型、电刺激的部位、参数, 动物点燃的行为表现、点燃方法及病理机制进行综述

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  • 强迫症脑成像研究进展

    强迫症是一种常见的、具有复发性和难治性的精神疾病,它以反复出现的强迫行为和强迫观念为主要特征。以往大量脑成像研究证实了强迫症患者存在皮质-纹状体-丘脑-皮质环路的异常,特别是眶额叶、扣带回、尾状核、丘脑、颞叶、脑岛、胼胝体等组织存在结构和功能异常。近年来的研究还发现强迫症患者的背外侧前额叶、顶叶、枕叶、小脑等脑区也存在着结构和功能障碍。此提示,强迫症患者可能还存在其他环路或系统的异常,就此进行探索有助于深入了解强迫症的病理机制。因此,该文尝试对近年来强迫症的脑结构和功能成像的研究进行综述,以期全面研究分析强迫症的神经生物学基础研究的最新动态,为临床诊断与治疗提供参考。

    Release date:2017-12-25 06:02 Export PDF Favorites Scan
  • Internal jugular vein insufficiency syndrome

    Long-term chronic internal jugular vein (IJV) insufficiency, originally viewed as a non-pathological finding, may result in cerebral venous outflow disturbance, leading to cerebral venous ischemia and cerebral nervous functional disorders. In this article we discuss probable etiologies, symptoms, diagnosis and treatment of IJV disturbance, so as to provide some insights for clinicians.

    Release date:2018-06-26 08:57 Export PDF Favorites Scan
  • Research progress on pancreatic cancer-associated diseases

    ObjectiveTo conclude the current status and research progress on the pathological mechanism, development and management of pancreatic cancer-associated diseases and provide evidence for intervention of such diseases.MethodThe relevant literatures were reviewed, and the research progress on pancreatic cancer-associated diseases were summarized.ResultsThere are many types of pancreatic cancer-associated diseases, and common diseases included pancreatic intraepithelial neoplasia, intraductal papillary mucinous neoplasm, mucinous cystic neoplasm, diabetes, and chronic pancreatitis. Although management and following-up about this kind of diseases remain controversial, the basic consensus has been reached.ConclusionAdequate follow-up is required for patients with pancreatic cancer-associated disease, individualized interventions should be taken if necessary.

    Release date:2020-07-01 01:12 Export PDF Favorites Scan
  • Research progress on the pathogenesis of ischemic bile duct injury after liver TACE

    ObjectiveTo summarize the research progress on the pathogenesis of ischemic bile duct injury after transcatheter arterial chemoembolization (TACE).MethodThe recent studies on the incidence, pathological features and related mechanisms of ischemic bile duct injury and ischemic bile duct injury after TACE were reviewed.ResultsThe incidence of ischemic bile duct injury after liver TACE fluctuated greatly and was related to different chemoembolization methods. At present, the causes of ischemic bile duct injury were attributed to the bile duct ischemia caused by embolization and the toxic effects of chemotherapeutic drugs. The destruction of protective mechanism of bile duct epithelium and the expression of transforming growth factor-β might play an important role in ischemic bile duct injury.ConclusionsAfter liver TACE, in addition to the direct injury of bile duct caused by the toxic effects of ischemia and chemotherapy drugs, the damage of bile duct epithelial protection mechanism caused by ischemia and chemotherapy drugs makes the toxic effects of bile acids play a very important role in the ischemic bile duct injury. However, there is still no direct evidence of bile duct epithelial protection mechanism in ischemic bile duct injury after liver TACE. Further clarifying the role of bile duct epithelial protection mechanism in ischemic bile duct injury after liver TACE will be helpful to explore its prevention and treatment measures, and provide new insights for the further studies in future.

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  • Case study—Typical imaging signs of epithelioid hemangioendothelioma

    This article presented readers with typical enhanced CT and MR images of a patient with epithelioid hemangioendothelioma, and briefly described the pathological mechanisms behind the typical imaging signs, in order to enhance the readers’ understanding and awareness of the typical imaging signs of this rare disease, and thus reduce its underdiagnosis rate and misdiagnosis rate.

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  • Research progress on pathological mechanism and clinical correlation between medial meniscus posterior root tear and tibial rotation

    Objective To summarize the current research progress on the concept, clinical presentation, diagnosis, biomechanical changes, and pathological mechanisms of the medial meniscus posterior root tear (MMPRT), and its clinical correlations with tibial rotation. MethodsThe research literature on MMPRT and its relationship with tibial rotation at home and abroad in recent years was extensively consulted and summarized. Results MMPRT is a specific and common type of medial meniscus injury of the knee joint. The occurrence of posterior medial pumping pain events following low-energy trauma in patients provides important clues for the diagnosis of this injury, with MRI being the preferred imaging modality. The biomechanical effects generated by MMPRT are similar to those caused by total removal of the medial meniscus. And this injury is usually associated with tibial rotation. MMPRT induces pathological external rotation of the tibia, which can be restored by timely medial meniscus posterior root repair. Furthermore, changes in tibial rotation are related to the healing status after medial meniscus posterior root repair. ConclusionMMPRT is closely related to tibial rotation. Understanding the biomechanics, pathological mechanisms, and clinical correlations between the two is of great significance for improving the diagnosis and treatment strategies.

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