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find Keyword "癫痫性脑病" 24 results
  • Advances in surgical treatment of early-infantile development epileptic encephalopathy

    Severe psychomotor developmental delay resulting from early postnatal (within 3 months) seizures can be diagnosed as Early-Infantile Developmental and Epileptic encephalopathies (EIDEE). Its primary etiologies include structural, hereditary, metabolic and etc. The main pathogenesis may be related to the inhibition of normal physiological activity of the brain by abnormal electrical activity and the damage of the brain neural network. Ohtahara syndrome and Early Myoclonic Encephalopathy (EME) are typical types of EIDEE. The principle of treatment is to improve the cognitive and developmental function by controlling frequent seizures. When the seizure is difficult to control with drugs, surgical evaluation should be performed as soon as possible, and surgical treatment is the first choice for patients suitable for surgery. The types of surgery can be divided into excision surgery, dissociation surgery, neuromodulation surgery and etc. The current status of surgical treatment of EIDEE was described, and the curative effect of surgical treatment was explored, so as to help clinicians choose appropriate treatment methods.

    Release date:2023-10-25 09:09 Export PDF Favorites Scan
  • RHOBTB2 基因变异所致发育性癫痫性脑病-64型一例并文献复习

    Release date:2024-11-20 10:50 Export PDF Favorites Scan
  • 吡哆醇(胺)氧化酶缺乏的研究进展

    吡哆醇(胺)氧化酶(Pyridox(am)ine-5-phosphate oxidase, PNPO)缺乏是一种由编码PNPO的PNPO基因突变引起的罕见常染色体隐性遗传的先天性代谢缺陷, 其典型临床表现是新生儿癫痫性脑病, 抗癫痫药物治疗无效, 磷酸吡哆醛或吡哆醇可改善症状, 目前尚无特异性生物化学标志物, 确诊需要检测PNPO基因。PNPO缺乏作为一种可治疗的潜在病因, 应包含在大田原综合征和新生儿肌阵挛性脑病的鉴别诊断中。未经磷酸吡哆醛或吡哆醇治疗的PNPO缺乏患儿可导致早期死亡, 而早期正确治疗者神经发育可正常。

    Release date:2016-10-02 06:51 Export PDF Favorites Scan
  • KCND3 新发突变引起的癫痫发育性脑病一例并文献复习

    Release date:2024-07-03 08:46 Export PDF Favorites Scan
  • 2023美国癫痫学会年会荟萃报道(三)

    美国癫痫学会(American Epilepsy Society,AES)年会是每年一度国际癫痫学界及工业界最受关注的会议。本年度的AES年会自2023年12月1日在奥兰多召开,为期5天,讨论了目前最受关注的癫痫学术领域及重点突破。本系列文章将分为五期,分别对大会每日的精彩内容进行荟萃报道:本文对大会第三日学术议程的内容进行了整理汇总,重点内容包括癫痫诊断及治疗的合理时机和最佳方案甄选,以及发育性癫痫性脑病、癫痫持续状态、基因性癫痫、术前评估、神经影像等热点话题。

    Release date:2024-03-07 01:49 Export PDF Favorites Scan
  • 突触融合蛋白结合蛋白-1 基因相关脑病的临床表型及基因研究进展

    癫痫性脑病是婴儿期和儿童早期严重的脑部疾患,其中 70% 癫痫性脑病与遗传因素相关。突触融合蛋白结合蛋白-1(Syntaxin-binding protein 1,STXBP1)基因编码 STXBP1,其发生突变可影响突触囊泡融合及神经递质释放,是引起癫痫性脑病的常见致病基因。STXBP1 基因突变的致病机制是单倍剂量不足,STXBP1 的补充或激活可能是一种潜在的精准治疗策略。文章对近年来报道的 STXBP1 基因相关脑病的临床表型、基因研究等进展进行综述。

    Release date:2018-05-22 02:14 Export PDF Favorites Scan
  • Dravet综合征——从癫痫性脑病到离子通道病

    编码电压门控钠通道α1亚单位(α1 subunit of the voltage gated sodium channel, SCN1A)的基因突变与多种癫痫综合征相关, 包括相对较轻的家族性全身性癫痫伴热性惊厥附加症(Genetic epilepsy with febrile seizures plus, GEFS+), 到严重的婴儿发病的Dravet综合征。已有证据显示, 大脑中不同神经元网络中SCN1A功能障碍的指向一个离子通道病模型, 使得Dravet综合征的神经科特征超越了单纯的发作相关损伤。基因改变的临床表型会随着其严重程度、个体的基因背景, 以及环境因素的不同而不同, 并且会根据离子通道的分布影响一系列神经元网络。原本就脆弱的系统可能很容易继发恶性事件, 如癫痫持续状态。离子通道模型预示着药物治疗并修复受损的γ-氨基丁酸(γ-aminobutyric acid, GABA)能神经传递也许不仅能预防癫痫发作, 还能治疗合并症。原综述研究关于在Dravet综合征中SCN1A突变的致病性, 及其对更广泛疾病表型的影响的最新证据, 并讨论了对这些特殊基因型的认识是否能影响临床实践。基因技术正在以前所未有的速度发展着, 将增加人们关于新的基因和基因间相互作用网络的知识。临床医生和遗传学家必将密切合作, 以保证基因测试结果能得到很好的解释和咨询服务

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  • Application of graph theory-based brain network in developmental and epileptic encephalopathy

    Developmental and epileptic encephalopathy (DEE) is a group of diseases that severely affects the neurological development of children, characterized by frequent seizures and significant neurodevelopmental impairments. These diseases not only impact the quality of life of affected children but also impose a heavy burden on families and society. In recent years, the development of brain network theory has provided a new perspective on understanding the pathological mechanisms of DEE, especially the role of structural and functional brain networks in the process of epilepsy. This review systematically summarized the research progress of structural and functional brain networks in DEE, highlighted their importance in seizure activity, disease progression, and prognosis evaluation.

    Release date:2025-01-11 02:34 Export PDF Favorites Scan
  • FGF13基因变异致发育性癫痫性脑病的临床及遗传学分析

    Release date:2025-05-08 09:41 Export PDF Favorites Scan
  • Clinical electrophysiological features of cyclin-dependent kinase-like 5 gene induced developmental epileptic encephalopathy

    ObjectiveTo investigate the clinical electrophysiological characteristics of Cyclin-dependent kinase-like 5 gene induced developmental epileptic encephalopathy (CDKL5-DEE). MethodsThe clinical data and series of video EEGs of children with CDKL5-associated developmental epileptic encephalopathy (CDKL5-DEE) who were admitted to the Children’s Medical Center of Peking University First Hospital from June 2016 to May 2024 were retrospectively analyzed. Results A total of 16 patients with CDKL5-DEE were enrolled, including 13 females and 3 males. All patients had de novo variants of CDKL5 gene, including 6 cases of missense variants, 5 cases of frameshift variants, 4 cases of nonsense variants, and 1 case of large fragment deletion. The age of onset was 8 days (d) after birth ~1 year (y) and 10 months (m), and the median age was (85.94±95.76) days. Types of seizures at onset: 4 cases of tonic seizures [age of onset 10~52 days, median age (25.5±15.84) days]; There were 5 cases of focal seizures [age of onset 8 d~8 m, median age (77.76±85.97) d]. There were 4 cases of epileptic spasmodic seizures [age of onset 3 m~1 y 10 m, median age (6.25±3.49) m]; There were 2 cases of bilateral tonic-clonic seizures [age of onset 30~40 days, median age (35.00±5.00) days]; focal concurrent epileptic spasm seizures 1 case (age of onset 2 m). A total of 59 VEEG sessions were performed in the pediatric EEG room of Peking University First Hospital for 4 hours. All the results were abnormal, including 26 normal background, 25 slow rhythm difference with background, and 8 no background. The interictal was 16 posterior or focal discharges, 19 multifocal discharges, 17 generalized or accompanied by focal/multifocal discharges, and 7 hypsarrhythmia; The ictal was 33 epileptic seizures, 6 myoclonic seizures, 5 focal seizures, 2 tonic-clonic seizures, 2 atypical absence seizures, 2 tonic seizures, 1 myoclonic sequential focal seizure, 1 focal sequential epileptic spasm, and 1 hypermotor-tonic-spasms. The background of patients within 6 months of age was normal, and the background abnormality increased significantly with age. generalized discharges are evident after 2 years of age between seizures. Conclusion CDKL5-DEE seizures have an early onset and are refractory to medications. Epileptic spasms are the most common type of seizure in every patient and long-lasting, with generalized seizures increasing markedly with age. EEG is characterized by a normal background within 6 months. With the increase of age, the background and interictal discharges have a tendency to deteriorate.

    Release date:2024-08-23 04:11 Export PDF Favorites Scan
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