【摘要】 目的 调查汶川地震伤残人员的日常生活活动能力(ADL),分析影响伤残人员日常生活活动能力的相关因素,为汶川地震伤残人员的社区康复提供依据。方法 利用ADL量表对283名汶川地震伤残人员的日常生活活动能力进行测量。结果 283名伤残人员地震伤残人员ADL平均得分为85.08±14.73,其中20.8%ADL完全正常,71.0%的伤残人员ADL日常生活能力轻度障碍,8.2%ADL中重度障碍；经多因素分析,地震伤残人员ADL的主要影响因素依次是:伤残程度、伤残合并疾病、伤残并发症、是否参与残疾人组织或相关活动(Plt;0.05)。结论 伤残人员ADL关键在于残疾程度,避免伤残并发症和二次残疾的发生,控制伤残合并疾病的发生,增加伤残人员的社会参与度,均有利于伤残人员ADL的提高。
Adenoid hypertrophy in children with epilepsy is rarely reported. This paper analyzes the clinical characteristics and incidence of adenoid hypertrophy in children with epilepsy.Methods The clinical data in children with epilepsy from December 2014 to April 2020 in Shenzhen Children's hospital were analyzed retrospectively.Results There were 449 cases diagnosed with adenoid hypertrophy (2.74%) in 16387 children with epilepsy. Among 449 cases of adenoid hypertrophy, 276 males (61.47%) and 173 females (38.53%). The age distribution was: 28 days to 1 year old, 8 cases (2%); 1-3 years old, 78 cases (17%); 3-6 years old 167 cases (37%); 6-12 years old, 153 cases (34%); 12-18 years old, 43 cases (10%). In 40 patients the IgG antibody were positive for EB capsid antigen in 25 (62.5%). In 56 cases of EB virus DNA were detected by fluorescence quantitative PCR, 25 (44.64%) positive, and 21/44 cases (47.72%) were positive by general nucleic acid detection of enteroviruses. The neutrophil reduction rate in peripheral blood was 42.19% in 673 tests, lymphocyteincreased in 292 (43.38%), platelet count increased in 307 (45.61%), abnormal in platelet hematocrit in 311 (46.21%); the mean volume of RBC was decreased in319 (47.39%) tests. The content of \begin{document}${\rm{HCO}_3^-} $\end{document} was reduced in 20/55 cases (36.36%). 25-hydroxy vitamin D was 33 (44.5%) decreased in 74 cases. The blood glucose was measured in 146 cases, 60 (41.09%) increased, total cholesterol was 31 (40.78%) increased in 76 cases, serum C peptide was 12 (29.26%) increased in 41 cases.Conclusion Adenoid hypertrophy in children with epilepsy may be related to infection, inflammation or immune disorder, which may cause nutritional, metabolic or internal environment disorders. Therefore, there is need of nursing and health education, transferring to specialized centers for diagnosis and treatment.