1. |
Coll RC, Holley CL, Schroder K. Mitochondrial DNA synthesis fuels NLRP3 inflammasome. Cell Res, 2018, 28(11): 1046-1047.
|
2. |
Qiu H, Liu W, Lan T, et al. Salvianolate reduces atrial fibrillation through suppressing atrial interstitial fibrosis by inhibiting TGF-beta1/Smad2/3 and TXNIP/NLRP3 inflammasome signaling pathways in post-MI rats. Phytomedicine, 2018, 51: 255-265.
|
3. |
Mao L, Kitani A, StroberW, et al. The role of NLRP3 and IL-1beta in the pathogenesis of inflammatory bowel disease. Front Immunol, 2018, 9: 2566.
|
4. |
Gutierrez Lopez J, Licata J, Pypker T, et al. Effects of heater wattage on sap flux density estimates using an improved tree-cut experiment. Tree Physiol, 2018 Dec 31.[Epub ahead of print].
|
5. |
Elliott EI, Sutterwala FS. Initiation and perpetuation of NLRP3 inflammasome activation and assembly. Immunol Rev, 2015, 265(1): 35-52.
|
6. |
Song N, Li T. Regulation of NLRP3 inflammasome by phosphorylation. Front Immunol, 2018, 9: 2305.
|
7. |
Hoss F, Rodriguez-Alcazar JF, Latz E. Assembly and regulation of ASC specks. Cell Mol Life Sci, 2017, 74(7): 1211-1229.
|
8. |
Shao BZ, Xu ZQ, Han BZ, et al. NLRP3 inflammasome and its inhibitors: a review. Front Pharmacol, 2015, 6: 262.
|
9. |
Artlett CM, Thacker JD. Molecular activation of the NLRP3 inflammasome in fibrosis: common threads linking divergent fibrogenic diseases. Antioxid Redox Signal, 2015, 22(13): 1162-1175.
|
10. |
Ren JD, Ma J, Hou J, et al. Hydrogen-rich saline inhibits NLRP3 inflammasome activation and attenuates experimental acute pancreatitis in mice. Mediators Inflamm, 2014, 2014: 930894.
|
11. |
Perregaux D, Gabel CA. Interleukin-1 beta maturation and release in response to ATP and nigericin. Evidence that potassium depletion mediated by these agents is a necessary and common feature of their activity. J Biol Chem, 1994, 269(21): 15195-15203.
|
12. |
Wang S, Yuan YH, Chen NH, et al. The mechanisms of NLRP3 inflammasome/pyroptosis activation and their role in Parkinson's disease. Int Immunopharmacol, 2019, 67: 458-464.
|
13. |
Sho T, Xu J. Role and mechanism of ROS scavengers in alleviating NLRP3-mediated inflammation. Biotechnol Appl Biochem, 2019, 66(1): 4-13.
|
14. |
Bordt EA, Polster BM. NADPH oxidase- and mitochondria-derived reactive oxygen species in proinflammatory microglial activation: a bipartisan affair? Free Radic Biol Med, 2014, 76: 34-46.
|
15. |
Man SM, Place DE, Kuriakose T, et al. Interferon-inducible guanylate-binding proteins at the interface of cell-autonomous immunity and inflammasome activation. J Leukoc Biol, 2017, 101(1): 143-150.
|
16. |
Yang J, Zhao Y, Shao F. Non-canonical activation of inflammatory caspases by cytosolic LPS in innate immunity. Curr Opin Immunol, 2015, 32: 78-83.
|
17. |
Gaidt MM, Ebert TS, Chauhan D, et al. Human monocytes engage an alternative inflammasome pathway. Immunity, 2016, 44(4): 833-846.
|
18. |
Lu B, Nakamura T, Inouye K, et al. Novel role of PKR in inflammasome activation and HMGB1 release. Nature, 2012, 488(7413): 670-674.
|
19. |
He Y, Franchi L, Nunez G. The protein kinase PKR is critical for LPS-induced iNOS production but dispensable for inflammasome activation in macrophages. Eur J Immunol, 2013, 43(5): 1147-1152.
|
20. |
Shenoy AR, Wellington DA, Kumar P, et al. MacMicking, GBP5 promotes NLRP3 inflammasome assembly and immunity in mammals. Science, 2012, 336(6080): 481-485.
|
21. |
Meunier E, Dick MS, Dreier RF, et al. Caspase-11 activation requires lysis of pathogen-containing vacuoles by IFN-induced GTPases. Nature, 2014, 509(7500): 366-370.
|
22. |
Shi H, Wang Y, Li X, et al. NLRP3 activation and mitosis are mutually exclusive events coordinated by NEK7, a new inflammasome component. Nat Immunol, 2016, 17(3): 250-258.
|
23. |
Schmid-Burgk JL, Chauhan D, Schmidt T, et al. A Genome-wide CRISPR (clustered regularly interspaced short palindromic repeats) screen identifies NEK7 as an essential component of NLRP3 inflammasome activation. J Biol Chem, 2016, 291(1): 103-109.
|
24. |
He Y, Zeng MY, Yang D, et al. NEK7 is an essential mediator of NLRP3 activation downstream of potassium efflux. Nature, 2016, 530(7590): 354-357.
|
25. |
Fry AM, O'Regan L, Sabir SR, et al. Cell cycle regulation by the NEK family of protein kinases. J Cell Sci, 2012, 125((Pt 19): 4423-4433.
|
26. |
Kanak MA, Shahbazov R, Yoshimatsu G, et al. A small molecule inhibitor of NFkappaB blocks ER stress and the NLRP3 inflammasome and prevents progression of pancreatitis. J Gastroenterol, 2017, 52(3): 352-365.
|
27. |
Aruna R, Geetha A, Suguna P. Rutin modulates ASC expression in NLRP3 inflammasome: a study in alcohol and cerulein-induced rat model of pancreatitis. Mol Cell Biochem, 2014, 396(1-2): 269-280.
|
28. |
尤运冬, 赵亮, 梅方超, 等. 巨噬细胞移动抑制因子抑制剂ISO-1在妊娠大鼠急性坏死性胰腺炎肠损伤中的作用. 中国普外基础与临床杂志, 2018, 25(11): 1308-1312.
|
29. |
武永胜, 李得溪, 赵海平, 等. 银杏叶提取物对重症急性胰腺炎大鼠脑组织中IL-1β、IL-6和TNF-α表达水平的影响. 中国普外基础与临床杂志, 2012, 19(6): 616-621.
|
30. |
Hoque R, Sohail M, Malik A, et al. TLR9 and the NLRP3 inflammasome link acinar cell death with inflammation in acute pancreatitis. Gastroenterology, 2011, 141(1): 358-369.
|
31. |
York JM, Castellanos KJ, Cabay RJ, et al. Inhibition of the nucleotide-binding domain, leucine-rich containing family, pyrin-domain containing 3 inflammasome reduces the severity of experimentally induced acute pancreatitis in obese mice. Transl Res, 2014, 164(4): 259-269.
|
32. |
Dong Z, Shang H, Chen YQ, et al. Sulforaphane protects pancreatic acinar cell injury by modulating Nrf2-mediated oxidative stress and NLRP3 inflammatory pathway. Oxid Med Cell Longev, 2016, 2016: 7864150.
|
33. |
Jakkampudi A, Jangala R, Reddy R, et al. Acinar injury and early cytokine response in human acute biliary pancreatitis. Sci Rep, 2017, 7(1): 15276.
|
34. |
Yu J, Ni L, Zhang X, et al. Surfactant protein D dampens lung injury by suppressing NLRP3 inflammasome activation and NF-kappaB signaling in acute pancreatitis. Shock, (2018).
|
35. |
Xu S, Wei S, Guo Y, et al. Involvement of nucleotide-binding and oligomerization domain-like receptors in the intestinal injury of severe acute pancreatitis in rats. Pancreas, 2018, 47(2): 245-251.
|