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find Author "颜艳" 5 results
  • 一种新型卧床患者支被架的设计与应用

    Release date:2017-07-21 03:43 Export PDF Favorites Scan
  • Application of Membrane Covered SelfExpanding Metallic Stent for 49 Patients with the Malignant Stricture of Advanced Esophagus and Cardia under Endoscope

    【摘要】目的探索内镜直视下应用带膜记忆合金支架对晚期食管、贲门恶性狭窄的介入治疗方法。方法2005年1月2009年11月对49例失去手术机会或拒绝手术的晚期食管癌或贲门癌致食管或贲门狭窄患者,行电子胃镜引导下放置镍钛合金支架;对狭窄程度重、胃镜不能通过者,先行Savary探条扩张再放置支架。结果49例均成功置入支架,解除狭窄有效率达100%。结论内镜直视下带膜支架置入操作方法简便、安全,可改善患者的生存质量,延长生存时间。

    Release date:2016-09-08 09:31 Export PDF Favorites Scan
  • Combined Therapy of Mosapride and Compound Digestive Enzymes for Functional Dyspepsia

    【摘要】 目的 观察莫沙必利联合复方消化酶胶囊治疗功 能性消化不良的临床疗效和安全性。方法 2008年1月—2009年7月对62例功能 性消化不良患者随机分为两组,每组31例,两组均口服莫沙必利片,三餐饭前30 min服用5 mg;试验组加服复方消化酶胶囊,进餐后30 min服用1粒。4周后观察疗 效。结果 试验组治疗总有效率明显优于对照组,分别为87.1%和61.3%(Plt;0 05)。对上腹痛、餐后饱胀、早饱、上腹烧灼感症状改善情况进行比较,试验 组治疗前后的症状总积分及症状改善有效率与对照组比较差异有统计学意义( Plt;005),而且起效时间明显缩短。两组患者均未发现严重不良反应。结论 莫沙必利联合复方消化酶胶囊治疗功能性消化不良安全有效,其疗效明显优于 单用促动力药莫沙必利。

    Release date:2016-09-08 09:37 Export PDF Favorites Scan
  • Arc调控神经元突触可塑性作用

    癫痫是常见的神经系统疾病之一,为脑部神经元高度同步化异常放电,其发病机制尚不明确。海马结构的苔藓纤维出芽和突触重塑学说是其形成的主要病理基础,也是癫痫长期、反复发作的重要原因。活性调节的细胞骨架蛋白(Activity regulated cytoskeletal protein,Arc)是一种谷氨酸神经元突触后细胞骨架相关蛋白,属于即刻早期基因,在脊椎动物中高度保守,被认为是参与突触重塑的重要因子。现将Arc的表达转录特征、Arc参与神经元细胞突触可塑性的结构性和功能性改变、突触可塑性参与海马苔藓纤维出芽诱发癫痫的发病、Arc通过调控海马神经元细胞突触可塑性及MFS参与癫痫的发病进行阐述,为研究Arc的突触可塑性作用为阐明癫痫致病机制提供新的方向和思路。

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  • RESEARCH PROGRESS OF REGULATION OF OSTEOCLAST FORMATION AND FUNCTION

    ObjectiveTo review the recent research progress of the regulatory mechanisms of osteoclast (OC) formation and functions. MethodsThe related literature on OC formation, activation, and functions was reviewed, analyzed, and summarized. ResultsMacrophage colony stimulating factor and receptor activator of nuclear factor-κB ligand are essential cytokines which regulate all aspects of OC formation and functions. In additional to mediating bone resorption, OCs also partici pate in regulation of osteoblast formation and immune responses. ConclusionThe researches on the regulation of OC formation and functions have further revealed the destruction mechanisms of various kinds of bone diseases, which will facil itate to find more suitable biological targets for cl inical therapy.

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